[1] NEUMANN E J, KLIEBENSTEIN J B, JOHNSON C D, et al.Assessment of the economic impact of porcine reproductive and respiratory syndrome on swine production in the United States[J].Journal of the American Veterinary Medical Association,2005,227(3):385-392. [2] ZHOU L, YANG H C.Porcine reproductive and respiratory syndrome in China[J].Virus Research,2010,154(1/2):31-37. [3] 郭宝清,陈章水,刘文兴,等.从疑似PRRS流产胎儿分离PRRSV的研究[J].中国畜禽传染病,1996(2):1-5. [4] KAPPES M A, FAABERG K S. PRRSV structure, replication and recombination:Origin of phenotype and genotype diversity[J].Virology,2015,479/480:475-486. [5] CHEN N H, XIAO Y Z, YE M X, et al.High genetic diversity of Chinese porcine reproductive and respiratory syndrome viruses from 2016 to 2019[J].Research in Veterinary Science,2020,131:38-42. [6] POPESCU L N, TRIBLE B R, CHEN N H, et al.GP5 of porcine reproductive and respiratory syndrome virus (PRRSV) as a target for homologous and broadly neutralizing antibodies[J].Veterinary Microbiology,2017,209:90-96. [7] HOU J, LI R, QIAO S L, et al.Glycoprotein 5 is cleaved by cathepsin E during porcine reproductive and respiratory syndrome virus membrane fusion[J].Journal of Virology,2020,94(10):e00097-20. [8] FERNÁNDEZ A, SUÁREZ P, CASTRO J M, et al. Characterization of regions in the GP5 protein of porcine reproductive and respiratory syndrome virus required to in duce apoptotic cell death[J].Virus Research,2002,83(1/2):103-118. [9] LIU C, NING Y B, XU B R, et al.Analysis of genetic variation of porcine reproductive and respiratory syndrome virus (PRRSV) isolates in central China[J].The Journal of Veterinary Medical Science,2016,78(4):641-648. [10] ROBINSON S R, ABRAHANTE J E, JOHNSON C R, et al.Purifying selection in porcine reproductive and respiratory syndrome virus ORF5a protein influences variation in envelope glycoprotein 5 glycosylation[J].Infection, Genetics and Evolution,2013,20:362-368. [11] 杨汉春. NADC30-like流行毒株重组毒株越来越多,不能用其他传染病的防控理念防控蓝耳病[J].北方牧业,2018(12):16. [12] 王瑜,贺仪,陈小洪,等.外来与新发动物疫病猪繁殖与呼吸综合征病毒NADC30-like变异流行情况及综合防控措施[J].猪业科学,2020,37(7):107-108. [13] 何奇松,孔子荣,李三木,等.广西猪源H9N2亚型流感病毒的分离与鉴定及HA、NA基因序列分析[J].黑龙江畜牧兽医,2021(5):80-84,91. [14] 周峰,常洪涛,赵军,等.2012—2013年猪繁殖与呼吸综合征病毒河南流行株的分离鉴定及分子流行病学调查[J].中国兽医学报,2014,34(9):1398-1404,1410. [15] 刘建奎,戴爱玲,杨小燕.NADC30-like PRRSV流行特点[J].中国猪业,2017,12(12):30-32,37. [16] 王政,解长占,于成东,等.广西PRRSV分离株Y12018全基因组扩增和遗传进化分析[J].中国兽医学报,2021,41(5):877-882. [17] 王景隆,全东群,莫清荣,等.一株类NADC30猪繁殖与呼吸综合征病毒的分离及遗传变异分析[J].中国动物传染病学报,2021,29(2):71-76. [18] SHI M, LAM T T Y, HON C C, et al. Phylogeny-based evolutionary, demographical, and geographical dissection of North American type 2 porcine reproductive and respiratory syndrome viruses[J].Journal of Virology,2010,84(17):8700-8711. [19] GUO Z H, CHEN X X, LI R, et al.The prevalent status and genetic diversity of porcine reproductive and respiratory syndrome virus in China:A molecular epidemiological perspective[J].Virology Journal,2018,15(1):2. [20] OSTROWSKI M, GALEOTA J A, JAR A M, et al.Identification of neutralizing and nonneutralizing epitopes in the porcine reproductive and respiratory syndrome virus GP5 ectodomain[J].Journal of Virology,2002,76(9):4241-4250. [21] 范玉凤,白娟,姜平.山东省猪繁殖与呼吸综合征病毒流行毒株GP5基因遗传变异分析[J].家畜生态学报,2017,38(4):63-67. [22] NI Y Y, OPRIESSNIG T, ZHOU L, et al.Attenuation of porcine reproductive and respiratory syndrome virus by molecular breeding of virus envelope genes from genetically divergent strains[J].Journal of Virology,2013,87(1):304-313. [23] VERHEIJE M H, OLSTHOORN R C L, KROESE M V, et al. Kissing interaction between 3′ noncoding and coding sequences is essential for porcine arterivirus RNA replication[J].Journal of Virology,2002,76(3):1521-1526. |